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Graduation Term

Spring 2026

Degree Name

Doctor of Philosophy (PhD)

Department

School of Biological Sciences

Committee Chair

Pirmin Nietlisbach

Committee Co-Chair

Alysia Vrailas-Mortimer

Committee Member

Jan Dahl

Committee Member

Tom Hammond

Committee Member

Ben Sadd

Committee Member

Nathan Mortimer

Abstract

PD is tightly linked with increased oxidative stress, which is thought to contribute to the death of the dopaminergic neurons leading to disease symptoms. Cells have evolved many different mechanisms to maintain redox balance and survive oxidative stress, including tightly regulated homeostasis pathways and antioxidant systems. Despite extensive study of oxidative stress responses, important gaps remain in understanding how these pathways are transcriptionally regulated and how the interact with genetic and environmental forms of PD. Here, we aim to address these gaps by 1) examining if disruptions in copper homeostasis are a risk factor for disease severity across genetic forms of PD and 2) characterizing how transcriptional regulation of p38Kb may contribute to an environmental induced form of PD using the paraquat model. Together, these studies aim to provide insight into the molecular mechanisms that underlie PD and potentially identify new therapeutic targets for the treatment of PD.

Access Type

Dissertation-ISU Access Only

Available for download on Saturday, July 15, 2028

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